The Origins of Health and Disease
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Fetal programming of body composition: relation between birth weight and body composition measured with dual-energy X-ray absorptiometry and anthropometric methods in older Englishmen. Am J Clin Nutr ; 82 5 —7. Developmental plasticity and human health. Nature ; — Developmental origins of health and disease: a paradigm for understanding disease etiology and prevention. Curr Opin Pediatr ; 27 2 — Maternal smoking during pregnancy predicts adult offspring cardiovascular risk factors—evidence from a community-based large birth cohort study.
PLoS One ; 7 7 :e Glucocorticoid exposure in late gestation permanently programs rat hepatic phosphoenolpyruvate carboxykinase and glucocorticoid receptor expression and causes glucose intolerance in adult offspring. J Clin Invest ; 10 — Prenatal glucocorticoid exposure leads to offspring hyperglycaemia in the rat: studies with the 11 b-hydroxysteroid dehydrogenase inhibitor carbenoxolone.
Diabetologia ; 39 11 — Prenatal hypoxia independent of undernutrition promotes molecular markers of insulin resistance in adult offspring. Programming of adult blood pressure by maternal protein restriction: role of nephrogenesis.
Developmental Origins of Health and Disease
Kidney Int ; 65 4 — Intrauterine programming of the endocrine pancreas. Diabetes, Obes Metab ; 9 s2 — Mechanisms underlying the developmental origins of disease. Rev Endocr Metab Disord ; 13 2 — Jaenisch R, Bird A. Epigenetic regulation of gene expression: how the genome integrates intrinsic and environmental signals. Nat Genet ; 33 3s — Persistent epigenetic differences associated with prenatal exposure to famine in humans.
Developmental Origins of Health and Disease | University of Helsinki
An unbalanced maternal diet in pregnancy associates with offspring epigenetic changes in genes controlling glucocorticoid action and foetal growth. Clin Endocrinol Oxf ; 77 6 — Programming of adipose tissue miR—3p and GDF-3 expression by maternal diet in type 2 diabetes. Cell Death Differ ; 19 6 — Mutations in the glucokinase gene of the fetus result in reduced birth weight.
Nat Genet ; 19 3 — DNA damage, cellular senescence and organismal ageing: causal or correlative? Nucleic Acids Res ; 35 22 — The intergenerational effects of fetal programming: non-genomic mechanisms for the inheritance of low birth weight and cardiovascular risk. J Endocrinol ; 1 :1— WHO Noncommunicable diseases; Moore SE. Early life nutritional programming of health and disease in the Gambia. J Dev Orig Health Dis ; 7 2 — WHO Global health risks; Developmental origins of health and disease: reducing the burden of chronic disease in the next generation.
Genome Med ; 2 2 WHO Preventing chronic diseases: a vital investment; Non-communicable diseases in low- and middle-income countries: context, determinants and health policy.
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I. HISTORY OF THE CONCEPTS
Stressed out? New research could explain why prenatal stress exposure increases vulnerability to stress later in life Pregnant women who experience stress, such as depression or anxiety, could be passing along a genetic marker for stress to their unborn fetus.
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